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Different methods may produce different results, and established treatment thresholds are limited
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These findings suggest that although GSH-dependent reductive metabolism supports cellular survival under physiological conditions, sustained mitochondrial activity enabled by GSH can become deleterious under stress (Fig

In idiopathic pulmonary fibrosis (IPF), the activation of AKT1 in alveolar macrophages is required for the development of pulmonary fibrosis [135]
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