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& Schmierer, K

Sprague et al, 2008)

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(2024): AIP Publishing

Obviously, further investigations are necessary to illustrate the pathological role of ferroptosis in AIH

When Glu release increases or reuptake is inhibited, continuous stimulation of NMDAR by Glu leads to massive calcium influx, resulting in excitotoxicity (Beas-Zarate et al., 2008)