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Analysis of the role of hepatic PPAR expression during mouse liver regeneration

Furthermore, whilst loss of C9orf72 in mice models disturbs microglial function, resulting in age-related neuroinflammation, this was not sufficient to cause neurodegeneration in C9orf72 knockout mice (Lall and Baloh, 2017)

These interactions highlight the multifaceted mechanisms by which polyphenols exert their therapeutic effects, targeting key pathological processes in AD, including oxidative stress, neuroinflammation, and synaptic dysfunction, thereby offering a promising avenue for therapeutic intervention

[DOI] [PubMed] [Google Scholar] 91.Hayano M., Yang W.S., Corn C.K., Pagano N.C., Stockwell B.R

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& Al-Harrass, M