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ATP7A-LINKED NEURODEGENERATIVE DISORDERS Unlike AT7B, where mutations in the gene result in a single known clinical phenotype (Wilson disease), mutations in ATP7A cause three X-linked neurodegenerative diseasesMenkes disease, occipital horn syndrome (OHS), and X-linked distal hereditary neuropathy (dHMN) (Kaler, 2011)

While estrogen provides protective benefits against skin ageing, it paradoxically promotes skin pigmentation by enhancing the activity of pigment-producing cells

No serious adverse events, clinically significant cardiovascular effects, or mutagenic, teratogenic, or fertility-related risks have been reported, supporting its favorable safety profile, including in pediatric populations ( Despite its promising antioxidant and immunomodulatory properties, several factors limit the rigorous evaluation of PYCs therapeutic potential

163 Chronic unpredictable stress (CUS) and acute stress both specifically reduce the expression of the gap junction-forming proteins Cx30 and Cx43, 164 and the integrity of the BBB is weakened in mice lacking Cx30 and Cx43