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Binding through the ARE, NAC also stimulates anti-inflammatory mechanisms via influencing hemoxygenase-1 (HO-1) or nicotinamide adenine dinucleotide phosphate (NADPH)-dehydrogenase-1 [44]

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Inflammatory eosinophils II showed loss of early activation markers and further intensified the expression of inflammatory genes, while inflammatory eosinophils III downregulated inflammatory genes and increased mitochondrial gene expression ( mt-Co3 , mt-Nd1 ), suggesting metabolic adaptation