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Alteration of such ratio during oxidative burst can modulate redox signaling pathways related to various cellular processes including proliferation, growth and apoptosis [1]

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We demonstrate how FOXO1 inhibition, or inhibition of KRAS G12D in SW48 +/G12D , results in the downregulation of glutamine synthetase

Moreover, the endogenous GSH pool is far too low to detoxify exogenous SF completely suggesting that reversible GSH conjugation is an inefficient detoxification mechanism in the presence of high ITC concentrations

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