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In vitro, hypoxia/reoxygenation models are typical, although not exactly equivalent to ischemic injury since lack of blood flow in vivo comes with other consequences

Journal of Dairy Science 82: 429443
Schutz gegen Stress-Ulzera) gezeigt

Waechtler, H

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Overall, the application of these neurons successfully models key pathological attributes that are characteristic of AD, including aggregation of amyloid beta, modified production of A40 or A42, hyperphosphorylation of TAU, reduction in synapses and their plasticity as well as amplification of oxidative stress [44]
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