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Upon binding to cytoplasmic glucocorticoid receptors (GR), corticosteroids induce translocation of GR complexes into the nucleus, where they interact with glucocorticoid response elements (GREs) to upregulate anti-inflammatory genes (e.g., IL-10, DUSP-1) and suppress pro-inflammatory mediators like IL-1, IL-6, TNF-, and chemokines, such as MCP-1 and IL-8 (McManus 2003

Glia, 60:1468-1480

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Maintaining proper concentrations of glycation products depends on a balance between, on the one hand, their endogenous formation and dietary intake, and on the other hand, the activity of antiglycation system and digestion of glycotoxins in the gastrointestinal tract

The fate of these substances, and the formation of chylomicrons, is illustrated by steps 1 to 8

CagriSema's NDA was filed with FDA in December 2025