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Implication GPX4 suppression, alone or with current TNBC therapies, impacts outcomes in preclinical TNBC models with or without obesity and offers a new, plausible mechanistic target for TNBC treatment
Downregulation of GSTM3 increased the cell proliferation capacity compared to shNC in PANC-1 stable cell lines (Figure 3A)
Clinical obesity trials tested 12-week cycles
The convenience of a capsule can sometimes lead to laxity
Understanding the physiological mechanisms of waterlogging stress during this stage is vital for formulating strategies to lessen its impact
doi: 10.1016/j.arr.2015.04.005
