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Description
Intestinal bacterial excess is more likely to happen in patients with Hashimotos thyroiditis, and studies suggest that HT is caused by modifications in intestinal flora, increased intestinal permeability and bacterial expansion

Yu et al
The other metric is flow rate

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Third, while we propose that HSL-derived fatty acids may serve as endogenous PPAR ligands, the direct identity of these ligands and the mechanism of NF-B suppression remain to be fully elucidated

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