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TMAO, in turn, can lead to epigenetic modifications as well as the formation of N-nitrous, which damage cellular DNA [43]

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Blockade of LCAC-mediated I Na(L) is also suggested to reduce the metabolic deficit developed with LCAC treatment (Wu et al., 2009)

273(32): 2037882 Wagner CA, Lukewille U, Kaltenbach S, et al

Shores NJ, Keeffe EB: Is Oral L: -Acyl-Carnitine an Effective Therapy for Hepatic Encephalopathy

The membrane was probed with antibodies against PPAR-, SOD1, CAT or -actin
