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EPIGENETIC REGULATORS AND THEIR METABOLIC FOOTPRINTS Beyond signal transduction drivers, mutational inactivation or neomorphic activation of epigenetic regulatory proteins contributes substantially to well-defined metabolic footprints that characterize specific AML subsets [9,27,28]

To further confirm this result, we treated tendon fibroblasts with BPC 157 at different concentrations (0, 0.1, 0.25 and 0.5 g/mL) for 24 h and the expression of growth hormone receptor was found to increase in a dose-dependent manner (Figure 1A,C)

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Food Chem (Oxf)