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128 In the context of mitochondrial diseases, genetically defective respiratory chain complexes or mitochondrial dysfunction can activate mt-ISR, which aims to enhance ATP production and antioxidant capacity, thereby attempting to restore mitochondrial homeostasis before mitophagy is activated

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On the other hand, the concentration of total thiols did not significantly increase, and catalase and GSH transferase (GT) activities decreased

Westergaard, E

10.1016/j.biocel.2017.05.015 152 UedaN.TakasawaK

The rationale behind this strategy is that pyridostigmine is a reversible carbamate inhibitor of acetylcholinesterase, which at doses used to treat myasthenia gravis, will occupy and protect a substantial percentage of acetylcholinesterase from being able to bind irreversibly with soman