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The interplay between calcium and iron metabolism is complex, as elevated intracellular calcium levels can further promote iron-induced cellular damage, creating a vicious cycle of injury in the myocardium (70, 71)

The cells treated for 1 h with 200 M of the ROS inducer TBHP were used as a positive control

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Moreover, 3,5-di-CQA shows inhibition of myocardial injuries through increasing the activity of phosphatidylinositol 3-kinase (PI3K)/Akt in tert -butyl hydroperoxide (TBHP)-treated H9C2 cells [96]

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