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It activates GLP-1R (class B GPCR) to stimulate Gs-cAMP-PKA signaling in pancreatic beta cells for glucose-dependent insulin secretion, suppress appetite through hypothalamic GLP-1R activation, and slow gastric emptying via vagal pathways

c FDA-approved for the treatment of type 2 diabetes and approved for chronic weight management in patients with obesity or overweight with at least one weight-related condition, mainly outside the USA

Note that cuprizone application [232] was an extremely high regimen and much higher than those commonly applied to mimic multiple sclerosis in rats [243,244])

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It is hypothesized to be linked to excessive MMP-1 activity, which could temporarily break down existing collagen faster than new collagen is built