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Journal of Neurochemistry 83, 1338-1348CrossRefGoogle ScholarPubMed 53Stocker, R., Yamamoto, Y., McDonagh, A.F
Bibiloni, R

Upon IB degradation, NF-B is released and translocates into the nucleus, where it recruits p300, which in complex with cAMP response element-binding protein acetylates RelA/p65, thereby activating NF-B transcription and enhancing the expression of downstream pro-inflammatory genes (Lawrence 2009)

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