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TRIM29 is uniquely and highly expressed in mouse alveolar macrophages (AMs) and degrades NEMO, thereby inhibiting the NF-B signaling pathway and reducing the production of pro-inflammatory cytokines, which plays a crucial role in maintaining immune homeostasis in the lungs (182)

Ideally incorporate rigorous baseline and follow-up assessments: DEXA, body composition, visceral fat, renal function, endocrine panels, inflammatory biomarkers (IL-6, TNF-, SASP markers if available)

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