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In summary, we introduce a programmed targeting nanosystem based on a sequentially responsive liposome to dual rectify the abnormal energy and stromal microenvironments via coordinated regulation of GLUT1 and NF-B signal in PDAC cells and PSC, thus improving the anti-cancer efficacy of PDAC

Circ-MBOAT2 knockdown represses tumor progression and glutamine catabolism by miR-433-3p/GOT1 axis in pancreatic cancer

[DOI] [PubMed] [Google Scholar] Serviddio G, Pereda J, Pallard FV, Carretero J, Borras C, Cutrin J, Vendemiale G, Poli G, Via J, Sastre J

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3e), indicating that the mutated Cys residues mediate the redox regulation of OsVIRK1 and are critical for its accumulation