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In summary, although our findings may discourage further investigation of ALCAR/ALA for treatment of bipolar depression, they should not discourage the study of other potentially beneficial mitochondrial-enhancing agents for bipolar disorderespecially maintenance treatment to address subthreshold mood symptoms

Additionally, LA may increase the efficiency of insulin signaling by regulating the expression and phosphorylation of IRS, subsequently decreasing hepatic lipid synthesis and storage

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This approach can extend battery life

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Chilliard Y., Toral P.G., Shingfield K.J., Rouel J., Leroux C., Bernard L., 2014