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This effect is further compounded by the inactivation of glutathione peroxidase 4 (GPX4) as ONOO can modify the selenocysteine residue within its active site, thereby disabling the only enzyme capable of directly reducing complex lipid hydroperoxides in biological membranes

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We treated hepatic and extra-hepatic (monocytic) cells with exosomes ALC/APAP

Fardin, A

doi: 10.2174/1570159X19666210712151556, 8