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Significantly higher frequencies of hematinic deficiencies and hyperhomocysteinemia in oral precancer patients

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Red blood cell formation and oxygen transport

The peptide promotes cell growth and tissue repair signals that could theoretically support cancer cell proliferation

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This finding is entirely consistent with results obtained previously for models of ferroptosis induced by erastin in SV40-transformed fibroblasts and BSO in fibroblasts from LHON patients ( Evidence for the contribution of mitochondrial iron-dependent oxidative events to ferroptosis was provided also by studies in which overexpression of the mitochondrial ferritin isoform (MtFt) was shown to inhibit ferroptosis induced by erastin ( In contrast to the effects of erastin, the use of FA allows the separation of mitochondrial lipid peroxidation and ferroptosis in fibroblasts