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2018 Oct 22;9:598

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This paper will discuss the effect of GHK on gene expression relevant to nervous system functions and cognitive decline as well as review genetic and laboratory data relevant to nerve outgrowth, copper transport into cells, anxiety and pain, DNA repair, the ubiquitin proteasome system, the anti-oxidant system, changes in gene expression for glial cells, astrocytes, brain cells, dendrites, ganglia, motor neurons, Schwann cells, and sensory cells

Together these results indicate that the succination of GSH, but not its detoxification, is responsible for the oxidative stress observed in Fh1-deficient cells GSH succination increases GSH biosynthesis We hypothesized that the chronic depletion of GSH caused by succination could perturb cell metabolism and contribute to a general deregulation of redox homeostasis

Superoxide dismutase 1 subunits with mutations linked to familial amyotrophic lateral sclerosis do not affect wild type subunit function

Il aide rparer l'ADN endommag et protger les cellules contre le stress oxydatif