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In APP/PS1 mice, BBB permeability and pericyte number are reduced, and -amyloid induces mitochondrial autophagy-dependent ferroptosis via the CD36/PINK/PARKIN pathway, resulting in blood-brain barrier damage and contributing to AD

Because of the absence of a nucleus and mitochondria, the red cell is incapable of generating energy via the (oxidative) Krebs cycle and depends mainly on the anaerobic conversion of glucose by the Embden-Meyerhof pathway (EMP, or direct glycolytic pathway) and the oxidative hexose monophosphate pathway (HMP, or pentose phosphate shunt) (Figure 23-1)

355,357,358,359 Dysfunction of the BBB includes disruption of BBB integrity (or BBB leakage), changes in BBB transport functions, reduced cerebral blood flow, and neuroinflammation

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Korner, M., Stockli, M., Waser, B