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In hereditary hemochromatosis, downregulation of the cystine-glutamate transporter directly exacerbates ferroptosis-driven liver injury, while N-acetylcysteine supplementation restores glutathione levels and inhibits ferroptotic cell death [54]

Despite the availability of various treatment options, many patients experience inadequate relief or intolerable side effects

A randomised controlled trial of a mitochondrial therapeutic target for bipolar depression: mitochondrial agents, N-acetylcysteine, and placebo

It has been observed that insulin resistance reduces mitochondrial respiration [56] and that human umbilical cord mesenchymal stromal cells from patients with GDM have premature senescence phenotypes and mitochondrial dysfunction

10.1002/nau.22965 [DOI] [PubMed] [Google Scholar] 100.Dias TR, Alves MG, Casal S, Silva BM, Oliveira PF