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glutathione inhibitor Vaccinia-related kinase 2 inhibition elicits vulnerability of metabolism in pancreatic cancer Glutathione-depleting self-immolative nanoparticles boost cuproptosis-driven

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In addition to cortical changes, structural alterations have been reported in subcortical regions, such as the putamina and thalami, independent of vascular abnormalities [22], while combined structural and functional abnormalities in the midbrain, thalamus and cerebellum further support the notion of widespread diabetes-related neural disruption in children [21]

glutathione inhibitor Vaccinia-related kinase 2 inhibition elicits vulnerability of metabolism in pancreatic cancer Glutathione-depleting self-immolative nanoparticles boost cuproptosis-driven

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glutathione inhibitor Vaccinia-related kinase 2 inhibition elicits vulnerability of metabolism in pancreatic cancer Glutathione-depleting self-immolative nanoparticles boost cuproptosis-driven

[DOI] [PMC free article] [PubMed] [Google Scholar] 95.Gervasio, F

glutathione inhibitor Vaccinia-related kinase 2 inhibition elicits vulnerability of metabolism in pancreatic cancer Glutathione-depleting self-immolative nanoparticles boost cuproptosis-driven

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glutathione inhibitor Vaccinia-related kinase 2 inhibition elicits vulnerability of metabolism in pancreatic cancer Glutathione-depleting self-immolative nanoparticles boost cuproptosis-driven

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glutathione inhibitor Vaccinia-related kinase 2 inhibition elicits vulnerability of metabolism in pancreatic cancer Glutathione-depleting self-immolative nanoparticles boost cuproptosis-driven

Atractylodin, the principal bioactive compound in Atractylodes macrocephala , possesses a range of pharmacological properties, including anti-inflammatory, antitumor, cardioprotective, and antioxidant activities (82)

glutathione inhibitor Vaccinia-related kinase 2 inhibition elicits vulnerability of metabolism in pancreatic cancer Glutathione-depleting self-immolative nanoparticles boost cuproptosis-driven

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