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Abstract Currently, all treatments for schizophrenia (SCZ) function primarily by blocking D 2 -type dopamine receptors
The acetylation of HSP90 affects its interaction with the critical co-chaperone p23, resulting in the loss of chaperone activity [42]
The primary pathway for osteoblast destruction via ROS involves mitochondrial apoptosis [16], which occurs through increased membrane permeability, release of cytochrome c, and apoptosis-inducing factor [17]
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Oral administration of -glutamylcysteine increases intracellular glutathione levels above homeostasis in a randomised human trial pilot study
