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SCD patients have aberrant metabolic regulation of fumarate, glutamate, glycine, malate, and other metabolites involved in glycolysis, nucleotide catabolism, and glutathione metabolism 3,4,5,6

This is consistent with the hypothesis that the cell has an over-capacity to chelate metal [208]

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When the concentration of Ca 2+ in the cytoplasm reaches a certain level, the calcium storage pool in the endoplasmic reticulum also releases Ca 2+ , inducing ERS (89, 90)

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Though a state of hyperammonemia in sepsis is not a hallmark, it is a sign of poor prognosis [48]