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Antiulcerogenic and Ulcer Healing Effects of Solanum nigrum (L.) on Experimental Ulcer Models: Possible Mechanism for the Inhibition of Acid Formation. Journal of Ethnopharmacology 104, no

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The increased inflammatory response exacerbates kidney damage in CKD patients, showing how chronic stress contributes to the progression of kidney disease through gut-derived signals (Zhou T

The difference between the effects of both QIs on the cell viability ( Figure 2 ) and caspase 3 activation ( Figure 4 ) suggests that upon GSH depletion, the decrease in cell viability is not only apoptosis dependent but also most likely depends other cell death pathways, such as necrosis ( IC 50 values of QIs under GSH-depleted conditions were around 20 M ( Figure 2 ), which is higher than the plasma concentrations of AQ and DEAQ found in patients, normally in nanomolar to micromolar ranges (Laurent et al., 1993

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