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doi: 10.1038/s41389-018-0098-7 123 PachecoRCiruelaFCasadVMallolJGallartTLluisCet al

miR-1246, released via exosomes from CRC cells, can reprogram tumor-associated macrophages (TAMs) toward a pro-tumor phenotype by altering their metabolic state, thereby supporting CRC growth and immune evasion [115]

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Finally, we summed the concentrations of 13 C or 15 N labeled metabolites in each replicate, which demonstrates that the added [ 13 C 5 15 N] glutamate is preserved in AMW50F and AMW50 + AOA, and is not lost but rather partitioned between glutamate, KG, and aspartate in AMW50 (Fig

The cancelled Phase I trial that would have tested oral BPC-157 in 42 healthy volunteers never published results, leaving that data inaccessible

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