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Lancet 398(10313):18111824

Marfella R, Sardu C, Balestrieri ML, et al

Individual calorie requirements on GLP-1 therapy depend on numerous physiological and lifestyle factors that must be considered for personalised nutritional planning

By combining the activities of GLP-1 and E2, we envisioned synergistic insulinotropic activities of these molecules on -cells

The process through which NAION destroys vision involves these sequential pathological events: Sudden blood supply disruption: Short posterior ciliary arteries feeding the optic nerve become blocked or experience severe flow reduction, starving nerve tissue Immediate nerve tissue death: Ischemic damage kills optic nerve cells within hours once dead, these neurons cannot regenerate Compartment syndrome: Initial swelling compresses remaining healthy nerve fibers in the confined scleral canal space, creating cascading damage Permanent structural changes: The optic nerve develops atrophy, with dead tissue replaced by glial scarring unable to transmit visual signals NAION commonly affects individuals with crowded optic discs or disc at risk, where cup-to-disc ratio measures 0.2 or less

However, some evidence suggests mechanisms involving glucose and insulin control, reducing body weight and BP, and improving blood lipid profiles [35]