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1970;19(Suppl):789-830
By engaging GLP-1 Rs across the gut-brain axis, these agents potentiate physiological satiety signals, slowing gastric emptying, suppressing hunger cues, and diminishing the hedonic appeal of calorie-dense foods
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91 In UC patients, endoplasmic reticulum function may be impaired, leading to abnormal accumulation of intracellular proteins and exacerbation of the cellular inflammatory response
